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Bactrim (Sulfamethoxazole trimethoprim)
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Bactrim

Generic Bactrim is a medication of sulfamethoxazole and trimethoprim antibiotics group. Generic Bactrim is used to treat: ear infections, urinary tract infections, bronchitis, traveler's diarrhea, Pneumocystis carinii pneumonia. Generic Bactrim fights against bacteria in your body.

Other names for this medication:
Actrim, Adrenol, Alfatrim, Altavit, Anitrim, Apo-bactotrim, Apo-sulfatrim, Assepium, Astrim, Avlotrin, Bacin, Bacsul, Bacta, Bactekod, Bactelan, Bacterol, Bacticel, Bactipront, Bactiver, Bactoprim, Bactramin, Bactricid, Bactricida, Bactrimel, Bactrizol, Bactron, Bactropin, Baktar, Baktimol, Bakton, Balkatrin, Balsoprim, Bascul, Berlocid, Betam, Bioprim, Biotrim, Biseptol, Biseptrin, Bismoral, Bitrim, Broncoflam, Bucktrygama, Cadaprim-r, Cadiprim, Canibioprim, Casicot, Chemitrim, Chevi-trim, Ciplin, Clotrimazol al, Co-sultrin, Co-trim, Co-trimoxazol, Co-try, Colizole, Comox, Cosat, Cotreich, Cotribene, Cotrim, Cotrimol, Cotrimox, Cotrimoxazol, Cotrimstada, Cotripharm, Cotrix, Cotrizol-g, Cots, Cozole, Daiphen, Danferane, Deprim, Dhatrin, Diatrim 24, Dientrin, Diseptyl, Ditrim, Doctrim, Dosulfin, Dotrim, Droxol, Drylin, Ectaprim, Editrim, Eliprim, Epitrim, Erphatrim, Esbesul, Escoprim, Eusaprim, Exazol, Feedmix ts, Fisat, Forcrim, Gantrisin, Gentrim, Globaxol, Groprim, Groseptol, Ifitrim, Ikaprim, Infatrim, Infectrim, Infectrin, Irgagen, Jasotrim, Kaftrim, Kanprim, Kemoprim, Kepinol, Kombitrim, Lagatrim, Lapikot, Letus, Licoprima, Linaris, Lupectrin, Medibiot, Megaset, Megatrim, Meprim, Methotrin, Methoxasol, Metoprim, Metoxiprim, Metrim, Momentol, Navatrim, Neoset, Neotrim, Netocur, Nopil, Novidrine, Novo-trimel, Novotrim, Noxaprim, Nu-cotrimox, Nufaprim, Octrim, Omsat, Onetrim, Organosol, Oribact, Oriprim, Ottoprim, Pehatrim, Pharex co-trimoxazole, Plocanmad, Politrim, Primadex, Primazol, Primazole, Primotren, Primsulfon, Purbac, Qiftrim, Regtin, Resprim, Ribatrim, Roxtrim, Sanprima, Sepmax, Septra, Septran, Septrin, Servitrim, Shatrim, Sigaprim, Sinatrim, Sinersul, Sitrim, Soltrim, Spectrem, Suftrex, Sulbron, Sulfa, Sulfagrand, Sulfamethoxazol, Sulfamethoxazole, Sulfamethoxazolum, Sulfametoxazol, Sulfatalpin, Sulfatrim, Sulfoid, Sulfoprima, Sulmetrim, Sulotrim, Sulphatrim, Sulphax, Sulphytrim, Sulprim, Sultri-c, Sultrian, Sultrim, Sultrima, Sumetoprin, Sumetrolim, Sunatrim, Suprasulf, Supreme, Suprim, Suprimass, Sutrim, Tabrol, Tagremin, Terasul-f, Terbosulfa, Theraprim, Tmps, Trelibec, Trifen, Triforam, Trim sulfa, Trima-kel, Trimaxazole, Trimecor, Trimesulf, Trimesulfin, Trimethazol, Trimethox, Trimetoger, Trimetoprim sulfa, Trimexazol, Trimexole-f, Trimezol, Trimidar-m, Trimoks, Trimol, Trimosazol, Trimosul, Trimoxsul, Trimsulint, Tripur, Trisolvat, Trisul, Trisulf, Trisulfose, Trisulin, Tritenk, Trizole, Two-septol, Urisept, Urobactrim, Vanadyl, Vanasulf, Wiatrim, Xepaprim, Yen kuang, Zaxol, Zoltrim

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Also known as:  Sulfamethoxazole trimethoprim.

Description

Generic Bactrim is taken to fight against ear infections, urinary tract infections, bronchitis, traveler's diarrhea, Pneumocystis carinii pneumonia. Generic Bactrim works by killing or slowing the growth of sensitive bacteria.

Generic Bactrim can't be given to children younger than 2 months old.

Bactrim is also known as Co-trimoxazole, Septra, Ciplin, Septrin.

Generic names of Generic Bactrim are Sulfamethoxazole, Trimethoprim.

Brand names of Generic Bactrim are Bactrim, Bactrim DS, Septra, Septra DS, Sulfatrim Pediatric.

Dosage

Generic Bactrim can be taken in tablets and liquid suspension.

Take Generic Bactrim orally.

Measure Generic Bactrim liquid suspension with a special dose-measuring spoon or cup, not a regular table spoon.

Use Generic Bactrim with full glass of water.

Generic Bactrim can't be given to children younger than 2 months old.

If you want to achieve most effective results do not stop taking Generic Bactrim suddenly.

Overdose

If you overdose Generic Bactrim and you don't feel good you should visit your doctor or health care provider immediately. Symptoms of Generic Bactrim overdosage: dizziness, drowsiness, nausea, vomiting, loss of appetite, stomach pain, headache, yellowing of your skin or eyes, blood in urine, fever, confusion, fainting.

Storage

Store at room temperature between 20 to 25 degrees C (68 to 77 degrees F) away from moisture, light and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Bactrim are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Do not take Generic Bactrim if you are allergic to Generic Bactrim components.

Do not take Generic Bactrim if you're pregnant or you plan to have a baby, or you are a nursing mother. Generic Bactrim can harm your baby.

Do not take Generic Bactrim if you have anemia.

Generic Bactrim can't be given to children younger than 2 months old.

Avoid exposure to sunlight, sunlamps, or tanning beds while taking Generic Bactrim.

Be careful with Generic Bactrim if you have kidney or liver disease, folic acid deficiency, asthma or severe allergies, AIDS, glucose-6-phosphate dehydrogenase deficiency (G6PD deficiency); if you are malnourished.

Be careful with Generic Bactrim if you take seizure medication such as phenytoin (Dilantin); diuretic (water pill); blood thinner such as warfarin (Coumadin); methotrexate (Trexall, Rheumatrex); methotrexate (Trexall, Rheumatrex); or an ACE inhibitor such as benazepril (Lotensin), captopril (Capoten), fosinopril (Monopril), enalapril (Vasotec), lisinopril (Prinivil, Zestril), moexipril (Univasc), perindopril (Aceon), quinapril (Accupril), ramipril (Altace) or trandolapril (Mavik).

It can be dangerous to stop Generic Bactrim taking suddenly.

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Tropheryma whippelii was found in the heart valves (three aortic valves and one mitral valve) of four patients with culture-negative endocarditis necessitating valve replacement. All patients had arthralgia for different lengths of time. Only one patient had mild gastrointestinal symptoms. Histologic characteristics of intestinal mucosa were normal in all patients, and polymerase chain reaction on intestinal biopsy was positive for T. whippelii in only one patient, who did not have diarrhea. In all patients, arthralgia resolved promptly after institution of antibiotic therapy. Disease did not recur in any patient after prolonged antibiotic therapy with cotrimoxazole.

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A high-performance liquid chromatography (HPLC) method was developed for the simultaneous analysis of trimethoprim (TMP), sulphamethoxazole (SMX), and acetylsulphamethoxazole (AcSMX) in small amounts of blood. The method involved precipitation with 50 microL trichloracetic acid (1M) to 125 microL plasma or serum sample. 60 microL supernatant was added to 60 microL mobile phase, modified with 50microL 1 M sodium hydroxide/mL. The mobile phase consisted of 20% acetonitrile and 80% phosphate buffer adjusted to pH 6.15. Using 125 microL of the sample, limits of quantitation were 0.1 microg/mL for TMP, 1.0 microg/mL for SMX, and 1.0 microg/mL for AcSMX. The precision of the method was 2% to 11% over the range of concentrations tested, 0.5-30 microg/mL for TMP, 5-300 microg/mL for SMX, and 2.5-150 microg/mL for AcSMX, respectively. No interference with other commonly used drugs was observed. The method is rapid, simple, specific, and sensitive enough for pharmacokinetic studies. The small amount of blood required makes it suitable for pediatric patients. The method was used to analyze samples from Tanzanian children aged 6-59 months participating in a cotrimoxazole (TMP/SMX)/chloroquine randomized trial for the treatment of uncomplicated malaria. Venous blood samples from 68 children were collected 2 hours after the first dose of TMP/SMX (4 mg/kg TMP/20 mg/kg SMX at two divided doses for 5 days) and again at treatment day 4. Individual variations in plasma concentrations of TMP, SMX, and AcSMX were considerable. The mean and SEM plasma concentrations (g/mL) of TMP, SMX, and AcSMX 2 hours after the first treatment dose were 2.0 +/- 1.0 (range 0.5-6), 53 +/- 22 (range 24-146), and 13.5 +/- 12 (range 0-65), respectively. On the fourth day the attained plasma concentrations were not significantly different from samples collected after the first dose.

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Between June 1988 and September 1996 12 of 65 infants (18%) admitted to the Department of Pediatrics, Ramathibodi Hospital, Bangkok, Thailand with purulent meningitis were infected with Salmonella spp. Their ages ranged from 1.5 to 6 months. Six of the infants had diarrhoea, 9 had seizures, and 11 had subdural effusion or empyema. Six infants required surgical treatment; 2 had brain abscesses. Salmonella was recovered from the cerebrospinal fluid of 11 infants and from the subdural fluid of 10. Eight infants were successfully treated with cefotaxime alone or in combination with co-trimoxazole, one with co-trimoxazole, and one with the combination of co-trimoxazole and ampicillin. The duration of treatment was 6 weeks, except for one patient who had a large brain abscess and was treated for 8 weeks. The last 2 patients, despite the fact that the organisms were susceptible to cefotaxime, failed to respond clinically to appropriate doses of it. Both were cured after ciprofloxacin was added to the therapy. Ciprofloxacin is probably the drug of choice to be used in addition to the previously used antibiotics for severe cases of Salmonella meningitis in infants.

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Thirty-five patients with meningitis caused by Haemophilus influenzae type b were admitted to our hospital from January 1981 to December 1984; 60% of the strains were resistant to ampicillin, 65.7% to chloramphenicol, and 57% to both antibiotics. No significant differences in age, sex, previous treatment, clinical symptoms, outcome, and mortality were found between the 20 patients whose strains were resistant to both ampicillin and chloramphenicol and the other 15 patients whose strains were susceptible to ampicillin, chloramphenicol, or both. Alternative therapeutic agents were a combination of carbenicillin and gentamicin and new cephalosporins. At present, cefotaxime is our treatment of choice for meningitis caused by H. influenzae type b.

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Streptococcus pneumoniae is a worldwide leading cause of morbidity and mortality, while susceptibility towards penicillin and macrolides can be less than 50% in many regions.

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This was a retrospective case-control study. Subjects were veterans with Gram-negative UTIs seen at the Philadelphia VA Medical Center from 1 July 1996 to 31 December 1999. Subjects were linked to a national VA outpatient pharmacy database. Cases and controls were identified based on the results of trimethoprim-sulfamethoxazole susceptibility testing.

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The purpose of this study was to determine whether gilthead sea bream and sea bass treated with combination of trimethoprim and sulfamethoxazole (TMP-SMX) differed in terms of physiological and innate immune biomarkers. Fish were exposed to TMP-SMX at 40 ppm concentration for 1 h as a prophylactic usage. Plasma cortisol, glucose, electrolytes (Ca, P, Na, K, Cl, and Mg) as well as plasma lysozyme activity, C-reactive protein (CRP), and ceruloplasmin (Cp) were measured soon after treatment and following 24 and 48 h in normal sea water for recovery. Treatment with TMP-SMX in both gilthead sea bream and sea bass led to an increase in plasma cortisol and glucose. Fluctuations in some electrolytes were found after treatment and during recovery period, however, the ratios of monovalent ions in treated sea bream were similar to control. Hematocrit values as well as plasma lysozyme activity in gilthead sea bream and sea bass were not affected by the treatment. CRP in gilthead sea bream and Cp in sea bass responded to the treatment with decreased levels. Both gilthead sea bream and sea bass displayed a rapid physiological stress response and sensitivity to TMP-SMX exposure, which requires more than 48-h period for regaining homeostasis.

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Patients received cyclosporine microemulsion or tacrolimus capsules twice/day in combination with once-daily sirolimus solution and corticosteroids. Sirolimus was administered 6 hours after the morning dose of cyclosporine or tacrolimus. After receiving a 6-mg loading dose of sirolimus, participants received sirolimus 2 mg/day for at least 7 days. Neither the cyclosporine nor the tacrolimus dosage was adjusted for at least 3 days before and during blood sampling for pharmacokinetic profiling.

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The records of 39 patients with CGD who were monitored during a period of 22 years were reviewed. All infections, infectious complications, and clinical outcomes were documented for a total observation period of 610 patient-years and were stratified with respect to different CGD subtypes.

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CTM has a good therapeutic effect in TL and may be used in selected patients for whom treatment is required.

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In a multicenter open study, intraperitoneal aztreonam was used together with vancomycin, cloxacillin, or flucloxacillin for initial empiric treatment of peritonitis associated with continuous ambulatory peritoneal dialysis (CAPD). Monotherapy with aztreonam was continued in 34 episodes of gram-negative peritonitis in 28 patients. The microorganisms isolated included Escherichia coli, Acinetobacter species, Pseudomonas species, and Klebsiella species. In three episodes, two organisms were cultured. Microbiologic assessment revealed cure in 27 episodes, cure with relapse in two, cure with superinfection in one, and treatment failure in four (with resistance to aztreonam in three). As assessed by clinical criteria, 27 episodes were cured, five failed to respond, and two responded partially. No adverse reactions to aztreonam were observed. Comparison of these 34 episodes with 35 episodes in historical controls (treated mainly with aminoglycosides and/or trimethoprim-sulfamethoxazole) showed significantly higher rates of cure (84% vs. 51%) and of survival (97% vs. 86%) as well as lower rates of catheter removal (65% vs. 87%) with aztreonam. Thus aztreonam is a safe and efficient agent for the treatment of peritonitis caused by gram-negative organisms in patients undergoing CAPD.

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The time-kill effect of moxifloxacin on 20 genetically distinct isolates of Stenotrophomonas maltophilia resistant to trimethoprim-sulfamethoxazole was studied. The majority (80%) were killed by a concentration equivalent to four times the MIC; the MIC induced a transient decrease in bacterial counts at 4 h, followed by regrowth. No effect was detected in four isolates. These results merit further clinical consideration.

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The authors report a case of macrocytic anemia due to folate deficiency occurring suddenly after the administration of trimethoprime-sulfamethoxazole and completely cured by folic acid. This type of complication occurs particularly often in patients who already have a relative folate deficiency. In our case only moderate alcoholism was found. Thus individual Buy Ondansetron Odt predisposition due to enzyme abnormality must be considered. Prophylactic administration of folic acid in patients receiving this drug association is thus advisable.

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To determine the association between trimethoprim-sulfamethoxazole (TMP-SMX) prophylaxis for Pneumocystis carinii pneumonia and risk of bacterial infections in persons with AIDS, we abstracted hospital records from 6496 adult admissions to 42 hospitals in western Washington state. Of these admissions, 570 involved 637 bacterial infections diagnosed among patients who had been prescribed prophylactic TMP-SMX or aerosolized pentamidine. Cases [admissions with bacteraemia, bacterial pneumonia, acute or chronic sinusitis, or urinary tract infection (UTI)] were compared to controls (admissions not associated with any of the 5 bacterial infections). After adjusting for CD4 lymphocyte count and presence of P. carinii pneumonia, TMP-SMX prophylaxis, relative to aerosolized pentamidine prophylaxis, was associated with a reduced risk of bacteraemia (adjusted OR = 0.5; 95% CI, 0.2-1.0; P = 0.04), bacterial Buy Hydroxyzine 10mg pneumonia (adjusted OR = 0.5; 95% CI, 0.3-0.8; P = 0.01), acute sinusitis (adjusted OR = 0.5; 95% CI, 0.2-1.3; P = 0.2), chronic sinusitis (adjusted OR = 0.3; 95% CI, 0.1-0.7; P = 0.01), and UTI (adjusted OR = 0.5; 95% CI, 0.2-1.2; P = 0.1), and all 5 bacterial infections combined (adjusted OR = 0.6; 95% CI, 0.5-0.8; P < 0.001).

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The cutaneous manifestations of human immunodeficiency virus infection include papulosquamous diseases, viral and fungal infections, and neoplastic disorders. Eczematous photosensitivity disorders have been reported in patients infected with the human immunodeficiency virus. We describe a patient with advanced acquired immunodeficiency syndrome who developed photodistributed hypertrophic lichen planus. We believe this is a Buy Prescription Nexium Online distinct cutaneous manifestation of human immunodeficiency virus infection.

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Compared with clindamycin Buy Zantac Liquid , use of trimethoprim-sulfamethoxazole or β-lactams was associated with increased risks of treatment failure and recurrence. Associations were stronger for those with a drainage procedure.

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Retrospective review of cases of Buy Tricorn nocardiosis in SOT at a large center from 2006 to 2012.

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To investigate the microbial etiology of suppurative chronic otitis media (SCOM) in patients with complete cleft lip and palate Buy Viagra Netherlands and isolated cleft palate and to determine the sensitivity of isolated microorganisms to antibiotics by drug diffusion from impregnated discs in agar and the minimum inhibitory concentration of each drug to these microorganisms by drug dilution in agar.

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A retrospective case note review of children admitted with serum sickness like reactions over a 10 year period was carried out. 59 children were Buy Zithromax 500mg identified and 30 of these were eligible for inclusion in the study.

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The bacteriological and clinical efficacy of norfloxacin 400 mg b.i.d. was compared to those of co-trimoxazole (160 mg of trimethoprim plus 800 mg of sulphamethoxazole) b.i.d. and placebo b.i.d. for the treatment of acute bacterial diarrhoea in a randomized double-blind trial. Of a total of 450 patients with acute diarrhoea, 303 had positive bacterial cultures and were evaluable for efficacy. The time to elimination of pathogens was significantly (p less than 0.001) shorter in the norfloxacin group than in the co-trimoxazole and placebo groups. At completion of treatment, bacteriological cure was found in 97.9%, 72.4% and 38.2% of patients treated with norfloxacin, co-trimoxazole and placebo, respectively. All pathogens were susceptible to norfloxacin and none of them developed resistance to norfloxacin during treatment. In the co-trimoxazole group, resistance to that antibiotic increased from 2% at inclusion to 65.6% at the end of treatment (p less than 0.001). In patients with shigellosis or cholera, the mean time to normalization of bowel movements was significantly shorter in the norfloxacin and co-trimoxazole groups than in the placebo group (p less than 0.05 and p less than 0.01, respectively). There were no significant differences between groups with respect to adverse events reported. In conclusion, norfloxacin was well tolerated and highly effective in the treatment of acute bacterial diarrhoea.