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Diflucan (Fluconazole)

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Diflucan is a high-quality medication which is taken in treatment of fungal infections, including yeast infections of the vagina, mouth, throat, abdomen, lungs, esophagus, blood, and other organs, meningitis caused by fungus, yeast infections in patients who are likely to become infected because they are being treated with chemotherapy or radiation therapy before a bone marrow transplant. It is working by slowing the growth of fungi that cause infection. It is triazole.

Other names for this medication:
Aflumicot, Afumix, Afungil, Albesin, Alfa flucon, Alozof, Anfasil, Azol-flucon, Batacan, Baten, Bagyne, Biskarz, Burnax, Byfluc, Candidin, Damicol, Dermyc, Diflazole, Diflazon, Diflu, Diflucozan, Difluzol, Difluzole, Difusel, Dikonazol, Dizole, Dizolo, Dofil, Duracan, Efac, Elazor, Exomax, Falipan, Farviron, Farzul, Felsol, Femixol, Figalol, Flanos, Flavona, Fluc, Fluc-hexal, Flucalit, Flucan, Flucomed, Flucon, Flucon-ac, Fluconal, Fluconamerck, Fluconapen, Fluconarl, Fluconax, Fluconazol, Flurit-g, Flusenil, Flutec, Fluval, Fluvin, Fluxes, Fluzol, Fluzole, Fluzomic, Fluzone, Forcan, Fugin, Fulkazil, Fultanzol, Govazol, Gynosant, Hadlinol, Honguil, Hurunal, Ibarin, Iluca, Kandizol, Kifluzol, Kinazole, Klaider, Klonazol, Lavisa, Lefunzol, Leucodar, Logican, Loitin, Lucan-r, Lucon, Lumen, Medoflucan, Medoflucon, Micoflu, Neofomiral, Nicoazolin, Nifurtox, Nispore, Nobzol, Nofluzone, Nor-fluozol, Novacan, Novoflon, Nurasel, Omastin, Opumyk, Oxifungol, Ozole, Plusgin, Ponaris, Proseda, Rarpefluc, Rifagen, Sacona, Sisfluzol, Stabilanol, Stalene, Sunvecon, Syscan, Ticamet, Tierlite, Tracofung, Trican, Triconal, Triflucan, Trizol, Unasem, Uzol, Varmec, Zemyc, Zenafluk, Zicinol, Zidonil, Zilrin, Zobru, Zolax, Zoldicam, Zolen, Zoloder, Zolstan, Zoltec, Zucon

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Azirol, Cymax, Levostat, Sporanox PulsePak, Sporanox, Noxafil, Nizoral, Mycelex Troche, Cresemba, Onmel, Oravig, Vfend


Also known as:  Fluconazole.


Diflucan is an effective remedy against fungal infections. Its target is to treat yeast infections of the vagina, mouth, throat, abdomen, lungs, esophagus, blood, and other organs, meningitis caused by fungus, yeast infections in patients who are likely to become infected because they are being treated with chemotherapy or radiation therapy before a bone marrow transplant.

Diflucan is working by slowing the growth of fungi that cause infection. It is triazole.

Diflucan is also known as Fluconazole, Forcan, Trican.

Generic name of Diflucan is Fluconazole.

Brand name of Diflucan is Diflucan.


Take Diflucan tablets and liquid form orally with or without food.

Do not crush or chew it.

Take Diflucan at the same time once a day with water.

If you want to achieve most effective results do not stop taking Diflucan suddenly.


If you overdose Diflucan and you don't feel good you should visit your doctor or health care provider immediately. Diflucan symptoms of overdosage: extreme fear that others are trying to harm you, hallucinations.


Store at room temperature below 30 degrees C (86 degrees F) away from moisture and heat. Throw away any medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Diflucan are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.


Do not take Diflucan if you are allergic to its components.

Do not take Diflucan if you're pregnant or you plan to have a baby, or you are a nursing mother.

Do not take cisapride (Propulsid).

Be careful with Diflucan if you are taking anticoagulants ('blood thinners') such as warfarin (Coumadin); astemizole (Hismanal) (not available in the United States); benzodiazepines such as midazolam (Versed); cyclosporine (Neoral, Sandimmune); disopyramide (Norpace); diuretics ('water pills') such as hydrochlorothiazide (HydroDIURIL, Microzide); erythromycin (E.E.S, E-Mycin, Erythrocin); isoniazid (INH, Nydrazid); moxifloxacin (Avelox); oral contraceptives (birth control pills); oral medicine for diabetes such as glipizide (Glucotrol), glyburide (Diabeta, Micronase, Glycron, others), and tolbutamide (Orinase); phenytoin (Dilantin); pimozide (Orap); procainamide (Procanbid, Pronestyl); quinidine (Quinidex); rifampin (Rifadin, Rimactane); sotalolol (Betapace); sparfloxacin (Zagam); tacrolimus (Prograf); terfenadine (Seldane)(not available in the United States); theophylline (TheoDur); thioridazine (Mellaril); valproic acid (Depakene, Depakote); and zidovudine (Retrovir), amiodarone (Cordarone); rifabutin (Mycobutin); dofetilide (Tikosyn).

Be careful with Diflucan if you suffer from or have a history of cancer, acquired immunodeficiency syndrome (AIDS), an irregular heartbeat, heart, kidney, liver disease.

Avoid alcohol.

Do not stop taking Diflucan suddenly.

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A 43-y-old woman with underlying cervix carcinoma stage IIIB, status postirradiation, and diabetes mellitus with tripathy suffered from chronic diarrhoea for more than 6 months. Stool examination showed few white blood cells and red blood cells. Rectal swab cultures disclosed Cryptococcus neoformans in 2 samples obtained 2 weeks apart. The diarrhoea responded to treatment with oral fluconazole for 4 weeks. Repeated rectal swabs revealed no yeast growth. During follow-up for 4 y, she had no problems with diarrhoea. Chronic diarrhoea caused by C. neoformans is rare and clinical suspicion is needed.

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Mucormycosis (or zygomycosis) is the term for infection caused by fungi of the order Mucorales. Mucoraceae may produce severe disease in susceptible individuals, notably patients with diabetes and leukemia. Rhinocerebral mucormycosis most commonly manifests itself in the setting of poorly controlled diabetes, especially with ketoacidosis.

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Two novel isolates of Candida glabrata exhibiting reduced sensitivity to amphotericin B (MIC, 8 μg ml(-1)) were found to be ERG2 mutants, wherein Δ(8)-sterol intermediates comprised >90% of the total cellular sterol fraction. Both harbored an alteration at Thr(121) in ERG2; the corresponding residue (Thr(119)) in Saccharomyces cerevisiae is essential for sterol Δ8-Δ7 isomerization. This constitutes the first report of C. glabrata harboring mutations in ERG2 and exhibiting reduced sensitivity to amphotericin B.

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The frequency of mucosal infections caused by Candida glabrata has increased significantly. Candida glabrata infections are often resistant to many azole antifungal agents, especially fluconazole. The purpose of this study was to compare the efficacies of posaconazole (PSC) and fluconazole (FLC) in the treatment of experimental C. glabrata vaginitis caused by isolates with different FLC susceptibilities. A battery of 36 vaginal isolates of C. glabrata was tested against PSC and FLC to determine their in vitro susceptibilities. The 48-h geometric mean MICs for all isolates tested were 0.156 and 4.238 μg ml(-1) for PSC and FLC respectively. Two strains of C. glabrata for which FLC MICs were different were selected for in vivo study. The treatment regimens for the vaginal murine infection model were PSC or FLC at 10 or 20 mg kg(-1) of body weight/day and 20 mg kg(-1) twice a day. Regimens with PSC at 20 mg kg(-1) once or twice a day were effective in reducing the load of both the FLC-susceptible and -resistant isolates of C. glabrata. FLC at 20 mg kg(-1) twice a day was effective in reducing the load of both the isolates of C. glabrata. PSC displayed a more effective in vivo activity than FLC in the treatment of murine C. glabrata vaginitis.

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Implant breast augmentation is one of the most frequently performed surgical procedures, and fungal infection still is considered exceptional. This report presents a case of bilateral breast implant infection by multidrug-resistant Candida albicans treated with a targeted antifungal therapy.

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Prophylaxis with intravenous fluconazole (400 mg per day) or placebo continued until resolution of the underlying surgical condition.

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Although the sensitivity and positive predictive value were low, the low mortality rate after fungal infection and the mild side effects of the preemptive treatment might justify our therapeutic strategy. Based on the effectiveness, this strategy warrants further investigation.

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Antiviral drug interactions are a particular problem among immuno-compromised patients because these patients are often receiving multiple different drugs, i.e. antiretroviral drugs and drugs effective against herpesvirus. The combination of zidovudine and other antiretroviral drugs with different adverse event profiles, such as didanosine, zalcitabine and lamivudine, appears to be well tolerated and no relevant pharmacokinetic interactions have been detected. The adverse effects of didanosine and zalcitabine (i.e. peripheral neuropathy and pancreatitis) should be taken into account when administering these drugs with other drugs with the same tolerability profile. Coadministration of zidovudine and ganciclovir should be avoided because of the high rate of haematological intolerance. In contrast, zidovudine and foscarnet have synergistic effect and no pharmacokinetic interaction has been detected. No major change in zidovudine pharmacokinetics was seen when the drug was combined with aciclovir, famciclovir or interferons. However, concomitant use of zidovudine and ribavirin is not advised. Although no pharmacokinetic interaction was documented when didanosine was first administered with intravenous ganciclovir, recent studies have shown that concentration of didanosine are increased by 50% or more when coadministered with intravenous or oral ganciclovir. The mechanism of this interaction has not been elucidated. Lack of pharmacokinetic interaction was demonstrated between foscarnet and didanosine or ganciclovir. Clinical trials have shown that zidovudine can be administered safely with paracetamol (acetaminophen), nonsteroidal anti-inflammatory drugs, oxazepam or codeine. Inhibition of zidovudine glucuronidation has been demonstrated with fluconazole, atovaquone, valproic acid (valproate sodium), methadone, probenecid and inosine pranobex; however, the clinical consequences of this have not been fully investigated. No interaction has been demonstrated with didanosine per se but care should be taken of interaction with the high pH buffer included in the tablet formulation. Drugs that need an acidic pH for absorption (ketoconazole, itraconazole but not fluconazole, dapsone, pyrimethamine) or those that can be chelated by the ions of the buffer (quinolones and tetracyclines) should be administered 2 hours before or 6 hours after didanosine. Very few interaction studies have been undertaken with other antiviral drugs. Coadministration of zalcitabine with the antacid 'Maalox' results in a reduction of its absorption. Dapsone does not influence the disposition of zalcitabine. Cotrimoxazole (trimethoprim-sulfamethoxazole) causes an increase in lamivudine concentrations by 43%. Saquinavir, delavirdine and atevirdine appeared to be metabolised by cytochrome P450 and interactions with enzyme inducers or inhibitors could be anticipated. Some studies showed that interferons can reduce drug metabolism but only a few studies have evaluated the pathways involved. Further studies are required to better understand the clinical consequences of drug interactions with antiviral drugs. Drug-drug interactions should be considered in addition to individual drug clinical benefits and safety profiles.

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When considering addition of a bioactive material to PMMA acrylic, Chlorhexidine will result in reduced fracture toughness of the acrylic base while Fluconazole has no effect.

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To examine the effect of fluconazole, a potent inhibitor of CYP2C9 and CYP2C19, on the pharmacokinetics of rosuvastatin in healthy volunteers. Significantly increased plasma concentrations of fluvastatin have been observed following co-administration with fluconazole.

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Prosthetic joint infections due to Cryptococcus neoformans have not been described before. We report a case of a prosthetic hip joint infection due to C. neoformans. An 84-year-old man with chronic lymphocytic leukemia presented with progressive left groin pain and fever. There was radiographic evidence of prosthesis loosening, and an aspirate of the left hip joint grew C. neoformans. The patient was treated with amphotericin B with good initial symptomatic response. The patient elected not to undergo revision arthroplasty, and oral suppressive therapy with fluconazole was initiated. After 10 months of fluconazole therapy, the prosthesis was removed secondary to pain and increased instability of the implant. Despite excellent penetration of fluconazole into the joint fluid and reports of successful outcome in patients with native joint cryptococcal infections Where To Buy Fexofenadine as well as prosthetic joint infections due to Candida spp., suppressive fluconazole therapy failed. The failure may have been due to an unfavorable interaction between the organism and immune mechanism as well as reduced activity of fluconazole in biofilm.

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The no-C. albicans and drug resistant strains to fluconazole and itraconazole can be found Buy Orlistat In Australia both in commensal and pathogenic oral Candida spp now.

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Two reviewers independently assessed trial eligibility, methodological quality and Buy Atarax Australia abstracted data.

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Esophageal disease is a common complication and cause of morbidity in patients with human immunodeficiency virus (HIV) infection. Buy Clomid In Mexico Opportunistic infections are the leading cause of esophageal complaints and may be a predictor of poor long-term prognosis, presumably as a reflection of severe underlying HIV immunodeficiency. The esophagus may be the site of the first acquired immunodeficiency syndrome (AIDS)-defining opportunistic illness in a large number of patients. Barium esophagography and upper gastrointestinal endoscopy are diagnostic modalities, commonly used to evaluate esophageal complaints in patients with AIDS. Treatment for most etiologies of esophagitis generally has a high degree of success, with a resultant improvement in quality of life. In addition to optimizing antiretroviral therapy, a thorough diagnostic assessment of every HIV-infected patient with esophageal complaints is warranted, followed by timely and appropriate treatment.

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This study showed that CgCDR1 gene was more closely related to fluconazole resistance of C. glabrata than CgCDR2 gene. In addition, several other genes related Buy Hydrochlorothiazide 50 Mg with fluconazole resistance of C. glabrata were identified.

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To review and update the incidence, mechanism, Buy Zantac For Dogs and clinical relevance of drug interactions with itraconazole, ketoconazole, and fluconazole.

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Three months after antifungal treatment, Ga-67 brain SPECT showed partial resolution of the lesion in the sella turcica region. The patient continued with fluconazole treatment for another 2 months and received another Ga-67 brain SPECT, which showed complete clearing of the previous lesion.

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Cryptococcus neoformans is the most common cryptococci causing infection in humans. Non-neoformans cryptococci have generally been regarded as saprophytes and rarely reported as human pathogens. We report a probable case of Cryptococcus laurentii meningitis in a HIV-infected patient and reviewed the literature on risk factors and treatment of this infection in humans. This patient was successfully treated with amphotericin B followed by fluconazole. Awareness of the emerging antifungal-resistant C. laurentii strains, as reported in the literature, should be emphasized, especially in immunocompromised patients.

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The results indicate that antifungal agents are efficient in the treatment of seborrheic dermatitis.

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Antifungal triazole agents (fluconazole, voriconazole, itraconazole and posaconazole) are widely used for the management of invasive fungal infections (IFI). These drugs are indicated both for the prophylaxis and treatment of IFI, particularly in candidiasis and aspergillosis, major cause of mortality in immunocompromised patients. Due to a large interindividual pharmacokinetic variability leading to sub-therapeutic or toxic concentrations and to concentration-efficacy and/or -toxicity relationships, therapeutic drug monitoring (TDM) of antifungal triazole is fully justified. This review provides an overview of literature based data that confirm the usefulness of such TDM and its level of evidence as well as the practical guidelines for its implementation. In addition, we discuss the interest of new tools to improve the clinical management of IFI, such as genotyping tests optimizing initial voriconazole dosing regimen or the development of a new solid oral tablet of posaconazole improving its bioavailability and limiting absorption disorders.