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Ponstel (Mefenamic Acid)
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Ponstel

Ponstel is in a group of drugs called nonsteroidal anti-inflammatory drugs (NSAIDs). Ponstel is used for treating menstrual pain. It may be used for short-term (not more than 7 days) treatment of mild to moderate pain. Ponstel blocks the effect of certain substances in the body that are associated with pain and inflammation.

Other names for this medication:
Acinic, Adsena, Aidol, Alfoxan, Algex, Algifemin, Algopress, Aprostal, Asimat, Bafhameritin-m, Beafemic, Benostan, Calmin, Cetalmic, Corstanal, Coslan, Dogesic, Dolarac, Dolfenal, Dolmetine, Fenamin, Fenamol, Fenaton, Fendol, Fensik, Flamic, Gardan, Gitaramin, Inflamyl, Laffed, Lapistan, Licostan, Lumental, Lysalgo, Mafepain, Masafen, Medicap, Mefac, Mefinter, Mefnac, Meftal, Meftan, Menin, Mephadolor, Molasic, Mycasaal, Namifen, Neuritorl c, Nichostan, Occorner, Omatan, Onemeday, Opistan, Pangesic, Parkemed, Pehastan, Pinalgesic, Ponac, Ponalar, Ponalgic, Poncofen, Pondex, Ponmel, Pontal, Pontalon, Pontin, Revalan, Rolan, Sicadol, Spiralgin, Sportusal, Stanalin, Tanston, Teamic, Topgesic, Tran-mf, Tynostan, Vidan, Youfenam

Similar Products:
Celebrex, Voltaren, Dolobid, Lodine, Motrin, Indocin, Orudis, Toradol, Naproxen, Ibuprofen, Diclofenac, Voltaren, Aleve, Advil, Celecoxib, Naprosyn, Motrin, Ketoprofen

 

Also known as:  Mefenamic Acid.

Description

Ponstel is used for treating menstrual pain. It may be used for short term (not more than 7 days) treatment of mild to moderate pain.

Ponstel blocks certain substances in the body that are linked to inflammation. NSAIDs treat the symptoms of pain and inflammation.

Ponstel is also known as Mefenamic acid, Ponstan.

Generic name of Ponstel is Mefenamic Acid.

Brand name of Ponstel is Ponstel.

Dosage

Take Ponstel orally.

Take Ponstel with or without food.

Take Ponstel with a full glass of water.

If you want to achieve most effective results do not stop taking Ponstel suddenly.

Overdose

If you overdose Ponstel and you don't feel good you should visit your doctor or health care provider immediately.

Storage

Store at room temperature between 15 and 30 degrees C (59 and 86 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Ponstel are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Do not take Ponstel if you are allergic to Ponstel components or to aspirin.

Do not take Ponstel if you are pregnant, planning to become pregnant, or are breast-feeding.

Do not take Ponstel if you have had a severe allergic reaction (e.g., severe rash, hives, trouble breathing, growths in the nose, dizziness) to aspirin or a nonsteroidal anti-inflammatory drug (NSAID) (e.g., ibuprofen, celecoxib).

Do not take Ponstel if you have had recent or will be having bypass heart surgery.

Do not take Ponstel if you have kidney problems.

Do not take Ponstel if you have ulcers or inflammation of the stomach or bowel.

Do not use Ponstel with aspirin.

Be careful with Ponstel when it is used by children younger than 14 years old and by elderly people.

Avoid machine driving.

Avoid drinking alcohol.

It can be dangerous to stop Ponstel taking suddenly.

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Local anaesthesia in addition to general anaesthesia can alleviate pain and distress caused by painful procedures in community dentistry for special needs patients. Further studies are needed to confirm these results in a larger group of patients.

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Forty patients with medication-resistant menorrhagia.

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A modified formalin test in mice was investigated. The pain response curve induced by 0.5% formalin was biphasic, having 2 peaks, from 0 to 5 min (first phase) and from 15 to 20 min (second phase). A low concentration of formalin was used, allowing the effects of weak analgesics to be detected. Centrally acting drugs such as narcotics inhibited both phases equally. Peripherally acting drugs such as aspirin, oxyphenbutazone, hydrocortisone and dexamethasone only inhibited the second phase. Aminopyrine and mefenamic acid which acted on both central and peripheral sites inhibited both phases, but the second phase was inhibited by lower doses. Thus, this method enables one to easily distinguish the site of action of analgesics. Furthermore, pain response in the first phase was inhibited by capsaicin-treated desensitization and Des-Arg9-(Leu8)-bradykinin (bradykinin inhibitor). The second phase was inhibited by compound 48/80 pretreatment, indomethacin and bradykinin inhibitor. Therefore, it is suggested that substance P and bradykinin participate in the manifestation of the first phase response, and histamine, serotonin, prostaglandin and bradykinin are involved in the second phase. These results indicate that the first and second phase responses induced by formalin have distinct characteristic properties, and it is a very useful method for examining pain, nociception and its modulation by pharmacological or other means.

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buy ponstel 2016-08-26

In serum-deprived G(o)-arrested cells, the addition of serum or growth factors initiates a cascade of events that culminates in DNA synthesis and mitosis. Recently, we showed that in mouse L-M(TK-) fibroblasts a 28-pS nonselective cation channel (NS channel) becomes quiescent at G(o Buy Nexium Singapore ) arrest and rapidly active within seconds of platelet-derived growth factor (PDGF) or serum addition, placing this response very early in the postreceptor signaling cascade. However, lack of specific channel blockers hindered determination of whether channel activation was necessary for mitogenesis. Derivatives of N-phenylanthranilic acid (DCA) have been reported to block a pancreatic nonselective channel. Therefore, using single-channel analysis, we examined the effect of these agents on the L-M(TK-) NS channel. Flufenamic acid and mefenamic acid rapidly produced reversible channel block with an inhibitory constant (Ki) approximately 10 microM. Furthermore, the component of the macroscopic K+ efflux shown to be mediated by the NS channel was blocked with a similar Ki value. DCA effects on cell proliferation were tested by measuring cloning efficiency and growth rate. Both were inhibited over the range of concentration that affected channel activity, and a 50% inhibitory dose of 50-100 microM was determined. This observation further substantiates the hypothesis that NS channel activation forms a necessary component in the transduction of the mitogenic signal from the PDGF receptor.

buy ponstel online 2015-04-03

In double-blind studies, mefenamic acid (group A) was compared with ibuprofen (group B) in the treatment of primary dysmenorrhea in 60 patients, during two cycles. The initial pain intensity was 8.9 for group A and 8.5 for B. The medication were administered at cero time (when the colic begin) and each 8 hours during the menstrual period. The decrease of pain intensity was presented after the second pill administration in group A for 8.6 + 1.8 at 6.0 + 2.4 in the first cycle and 8.5 +/- 1.8 at 5.9 +/- 2.6 in the second cycle. The group B the decrease of pain was similar of 8.2 Buy Cat Inhalers +/- 1.7 at 5.1 +/- 2.6 for the first cycle and the 8.2 +/- 1.7 at 4.7 +/- 2.6 in the second cycle of treatment. The number of tablets administered for both groups were for group A 5.5 and for B 4.4. The duration of pain were 22.1 hours for group A and 19.3 for B.

where to buy ponstel 2016-05-29

The aim of this study was to determine in vitro the potential of Aloe Vera juice as a skin permeation enhancer; a secondary aim was to probe the extent to which Aloe Vera itself permeates the skin. Saturated solutions of caffeine, colchicine, mefenamic acid, oxybutynin, and quinine were prepared at 32 degrees C in Aloe Vera juice and water (control) and used to dose porcine ear skin mounted in Franz diffusion cells with water as receptor phase. Receptor phase samples were taken over a 48 h period and permeants determined by reverse-phase HPLC. For caffeine and mefenamic acid no significant enhancements occurred between Aloe Vera and water as vehicles (p>0.05). However, for colchicine, oxybutynin and quinine the presence of Aloe Vera within the formulation provided enhancements (p < or = 0.05). Enhancement potential was dependent upon the molecular weight of the drug in formulation, with the enhancement effect attributable to as yet unidentified components within the Aloe Vera. Colchicine, with a molecular weight of 399.44, achieved the best enhancement with an enhancement ratio of 10.97. No correlation with lipophilicity was apparent. In a further experiment, where freeze-dried Aloe Vera was reconstituted at 200% residue level, permeation of quinine was 2.8 x that from normal Aloe Vera, providing further evidence for the presence of an enhancing factor within Aloe Vera. Certain, although unidentified, components of Aloe Vera readily permeated skin and the relative amount by which they permeated skin was inversely related to the molecular weight of the drug in solution, thus enhancement ratio. A new mechanistic rationale is proposed whereby larger drug solutes inhibit the permeation of Aloe Vera components, but also are then able to interact Buy Losartan more effectively with the enhancing factor and be subject to the pull effect.